Reactive
Attachment Disorder and Attachment
Disorders
At
the extreme end of the attachment disorders continuum is Reactive
Attachment Disorder (RAD). Attachment can vary from normal (or healthy)
on one end of the continuum, to mild or weak on the other end, to
extreme end as characterized by RAD. In the figure below, "Issues"
refer to that area where your teen or child has attachment issues but
not severe enough to fall into the RAD range.
Attachment
refers to the ability to form healthy relationships. These children
(teens, adults) have difficulty forming developmentally appropriate
social relationships.
Most often a problem with adopted
children, attachment problems can be brought about by trauma, abuse, or
neglect. In general, attachment problems begin because of
a lack of
healthy attachment to the primary care giver in the first two years of
life.
Healthy attachment
Healthy attachment is
characterized by emotional security and trust in relationships. For the
new born baby there is a "dance" of emotional attunement that occurs
between mother and child.
It begins with the mother-child relationship that extends
through
the first four years of life, with the first two years being extremely
critical. This dance involves the responsiveness of mother and child to
non-verbal and verbal signaling as the baby learns to recognize
emotions and their meanings, is loved, and nurtured.
As the
child grows, he or she learns to break away from the mother and to
explore the environment around him. Returning back to his mother's
safety for reassurance. In this way the foundation for individuation
and healty independence is laid as well as intimacy.
Weak attachment
Weak attachment is indicated by the child that
needs to control the adults around them,
especially the emotions and behaviors of caregivers
continually engages in power struggles and is
compulsive about winning them
repeatedly say "No" as a means of empowering
themselves
cause emotional and/or physical pain to others
maintain a negative concept of self
limited ability to regulate their emotions
(affect)
avoid mutually give-and-take games, laughter,
and fun with others
seem determined not to ask for help or to need
others
negatively reacts to praise, love, attempts to
make them feel special or worthy
One
of the adolescents, age 12, with which we worked delighted in
emotionally abusing his peers, was incredibly controlling with others,
including his parents, and could care less about relations with others.
He had minimal empathy. His parents could come or
go. It made little difference to him. His world was completely
ego-centric. AJ was a blooming personality disorder in the making with
a scary mix anti-social, histrionic, and narcassistic traits.
Toward
the end of our therapeutic relationship he was aware of how he affected
others and his total selfishness. He was brutally honest about himself
and his motivation. The best we could do in therapy was to make him
aware of how his behaviors and words affected others so that at least
he could make conscious choices about his manipulations and actions.
We
wished there could have been a happy ending to AJ's story. But the
clinical truth seems to be that after 10 years of age or so, the
attachment "damage" is not ameanable to therapy. This may because there
have been permanent changes in the teen's brain that are irreversable.
Neurological Changes
Research
indicates that normal attachment may be required for optimal brain
development (Schore, 2002). Again, weak attachment appears to be caused
by trauma, abuse,
or neglect during infancy or early childhood. These traumatic
attachments result in neurological imprinting which involves the
developing limbic and the autonomic nervous systems. Such imprinting
results in permanent changes in the neural pathways and
neurophysiology.
There
is increasing suspicion that it is the mirror neuron system that is
affected
(Rajmohan and Mohandas. 2007) as mirror neurons play important roles in
bonding and empathy.
RAD
The Diagnostic and
Statistical Manual IV R gives the following symptoms for
Reactive Attachment Disorder of Infancy or Early Childhood:
A.
Markedly disturbed and developmentally inappropriate social relatedness
in most contexts, beginning before age 5 years, as evidenced by either,
1. persistent
failure to
initiate or respond in a developmentally appropriate fashion to most
social interactions, as manifest by excessively inhibited,
hypervigilent, or highly ambivalent and contradictory responses (e.g.
the child may respond to caregivers with a mixture of approach,
avoidance, and resistance to comforting, or may exhibit frozen
watchfulness)
2. diffuse attachements as manifest by indiscriminate
sociability with marked inability to exhibit appropriate selective
attachments (e.g., excessive familiarity with relative strangers or
lack of selectivity in choice of attachment figures)
B.
The disturbance in Criterion A is not accounted for solely by
developmental delay (as in Mental Retardation) and does not meet
criteria for a Pervasive Developmental Disorder.
C. Pathogenic care as evidenced by at least one of the following:
1. persistent
disregard of the child's basic emotional needs for comfort,
stimulation, and affection
2. persistent disregard of the child's basic physical needs
3. repeated changes of primary caregiver that prevents formation of
stable attachments (e.g., frequent changes in foster care)
D.
There is a presumption that the are in Criterion C is responsible for
the disturbed behavior in Criterion A (e.g., the disturbance in
Criterion A began following the pathogenic care in Criterion C).
There are two subtypes. Inhibited
Type with the predominant disturbance is the persistent
inability to form healthy and appropriate attachments. Disinhibited Type in
which there is indiscriminate and inappropriate attachments.
Traditional psychotherapeutic approaches are ineffective with these
children and teens. Many can attach better to animals than humans. In
another case, for example, the client was more attached to her pet dog
than to the rest of her family. This ability to attach somewhat to
animals can be a starting point for such teens and children. Equine
Assisted Psychotherapy is useful in this regard.
As
emphasized in AJ's story above, beyond the age of about 10 or so years,
most clinicians, if they are honest about it, have little success in
facilitating major changes with attachment disorders
because of the imprinting
aspect. That is, the changes seem to be hard-wired into the neuronal
circuitry by that time.
Daniel Hughes in his book, Facilitating
Developmental Attachment,
presents an integrative psychotherapeutic approach which combines
Ericksonian Utilization theory, emotional engagement in spite of
conflicts and resistance, focusing on the natural attachment sequence
(feelings of closeness to mother, experiencing a break in the
relationship, followed by re-estabilishing the relationship). It relies
very much on relationship building between therapist and child and
between child and parent in the therapy sessions. He include physical
contact with the therapist in the form of holding and caresses. This
works well for young children but not so much for teens. He
also utilizes psychodrama in parents, child and therapist play
different roles.
Unfortunately his book is directed toward adopted and younger children.
However, there is a lot of good material in his book that applies also
to teens. Especially note his chapters for parenting these children and
teens. For more details on this and treatment strategies, we refer you
to his book:
Hughes, Daniel. 2004. Facilitating
Developmental Attachment: The Road to Emotional Recovery and Behavioral
Change in Foster and Adopted Children. Rowman &
Littlefield Publishers NY. 264 pp. Rajmohan V, Mohandas E. 2007 Mirror neuron
system. Indian J
Psychiatry [serial online] [cited 2008 Dec
15];49:66-9. Available from:
http://www.indianjpsychiatry.org/text.asp?2007/49/1/66/31522
Schore,
A. N. 2002. Dysregulation of the right brain: a fundamental
mechanism of traumatic attachment and the psychopathogenesis of
posttraumatic stress disorder. Australian
and New Zealand Journal of Psychiatry 36 (1): 9-30.